Premature aging-like phenotype in fibroblast growth factor 23 null mice is a vitamin D-mediated process

Mohammed S. Razzaque, Despina Sitara, Takashi Taguchi, René St-Arnaud, Beate Lanske

Research output: Contribution to journalArticle

Original languageEnglish (US)
Pages (from-to)720-722
Number of pages3
JournalFASEB Journal
Volume20
Issue number6
DOIs
StatePublished - Apr 2006

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Gene expression regulation
25-Hydroxyvitamin D3 1-alpha-Hydroxylase
lymphatic system
fibroblast growth factors
T-cells
Fibroblast Growth Factors
gene deletion
Glucuronidase
gene expression regulation
Cell proliferation
vitamin D
atherosclerosis
Vitamin D
cell proliferation
Skin
Animals
intestines
T-lymphocytes
Aging of materials
lungs

ASJC Scopus subject areas

  • Agricultural and Biological Sciences (miscellaneous)
  • Biochemistry, Genetics and Molecular Biology(all)
  • Biochemistry
  • Cell Biology

Cite this

Premature aging-like phenotype in fibroblast growth factor 23 null mice is a vitamin D-mediated process. / Razzaque, Mohammed S.; Sitara, Despina; Taguchi, Takashi; St-Arnaud, René; Lanske, Beate.

In: FASEB Journal, Vol. 20, No. 6, 04.2006, p. 720-722.

Research output: Contribution to journalArticle

Razzaque, Mohammed S. ; Sitara, Despina ; Taguchi, Takashi ; St-Arnaud, René ; Lanske, Beate. / Premature aging-like phenotype in fibroblast growth factor 23 null mice is a vitamin D-mediated process. In: FASEB Journal. 2006 ; Vol. 20, No. 6. pp. 720-722.
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